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Friday, July 6, 2012

Alexey Adamov, the painter. Painting: Landscape, genre, sea (marine).





About the artist
About Alexei Adamov it is not easy to tell all because a true artist is always in search. It is well-known search that assumes a difficult or even intricate road in life. After all, life is not a bed of roses and no roses without thorns. Naturally, you can disagree with the painter’s point of view, but an instinct prompts us to agree — we feel unable to be indifferent to Adamov’s painting. It contains the charm and fascination of contemplating the beauty of nature. And it sets us thinking. First of all, it sets thinking about eternity and momentariness of human existence, about the joy of life and sadness of wasting away. It is impossible to simulate all these emotions. It is possible only to come nearer to them little by little, enriching one’s divine gift of aesthetic vision.


Alexei Adamov is a person of indomitable energy. After his first personal exhibition of paintings in Taganrog in the building of Chekhov Library in 1997, Adamov became a well-known painter: his exhibition at the Don Artistic Fund in 2001, in Prague in the years of 2001-2002, in 2003 his pictures were exhibited in Paris and in Moscow Exhibition Hall of the Union of Artists. Adamov’s works of art constantly take part in opening days and exhibitions in various galleries of Moscow and of Europe. On August 13, 2004 was held his exhibition at the Moscow Exhibition Hall of the Union of Artists. This fact is undoubtedly proof of his innate genius and great acknowledgement of his creative personality.


You know Art is Beauty and it is a sign of kindness, too. We hope this eternal value won’t be depleted by the generous artist.


Ms.Ludmila Milukova






























































































































































































































































Boston College Chemists use nanowires to power photosynthesis




In this study, Boston College chemists use nanowires to power photosynthesis - collecting sunlight energy that powers reactions capable of synthesizing basic compounds of two popular pain-killing, and anti-inflammatory drugs.

Chemists use nanowires to power photosynthesis
NIST silicon nanowires. A nanowire is a wire of dimensions the size of a nanometer (a billionth of a meter). Alternatively, nanowires can be defined as structures that have a lateral size constrained to tens of nanometers or less and an unconstrained longitudinal size. (Photo: Softpedia)
Harnessing the power of the sun has inspired scientists and engineers to look for ways to turn sunlight into clean energy to heat houses, fuel factories and power devices. While a majority of this research focuses on energy production, some researchers are looking at the potential uses of these novel solar technologies in other areas.
Boston College Assistant Professor of Chemistry Dunwei Wang’s work with silicon nanowires and his related construct, Nanonets, has shown these stable, tiny wire-like structures can be used in processes ranging from energy collection to hydrogen-generating water-splitting.
Teaming up with fellow Boston College Assistant Professor of Chemistry Kian L. Tan, the researchers have taken aim at a role for nanowires in photosynthesis.
Their work has produced a process that closely resembles photosynthesis, employing silicon nanowires to collect light energy to power reactions capable of synthesizing the basic compounds of two popular pain-killing, anti-inflammatory drugs, they report in the current edition of Angewandte Chemie, the journal of the German Chemical Society.
The reaction sequence offers an approach that differs from earlier attempts to sequester carbon dioxide with sunlight and solves the vexing problem of carbon’s low selectivity, which so far has limited earlier methods to the production of fuels. Tan and Wang report their process offers the selectivity required to produce complex organic intermediaries capable of developing pharmaceuticals and high-value chemicals.
The process succeeds in taming stubborn carbon, which structurally resists most efforts to harness it for a single chemical product. Typically, refined forms of carbon molecules must first be produced to produce the necessary results.
“If we can start to use carbon dioxide and light to power reactions in organic chemistry, there’s a huge benefit to that. It allows you to bypass the middle man of fossil fuels by using light to drive the chemical reaction,” said Tan. “The key is the interaction of two fields – materials and synthetic chemistry. Separately, these fields may not have accomplished this on their own. But together, we combined our knowledge to make it work.”
During photosynthesis, plants capture sunlight and use this solar energy and carbon dioxide to fuel chemical reactions.
Tan and Wang used silicon nanowires as a photocathode, exploiting the wire’s efficient means of converting solar energy to electrical energy. Electrons released from the atoms in the nanowires are then transferred to organic molecules to trigger chemical reactions.
In this case, the researchers used aromatic ketones, which when struck by electrons become active and attack and bind carbon dioxide. Further steps produced an acid that allowed the team to create the precursors to ibuprofen and naproxen with high selectivity and high yield, the team reports.
Tan and Wang were joined in the research by Research Assistant Guangbi Yuan, PhD ’12, graduate student Rui Liu, doctoral student Candice L. Joe, and former doctoral student Thomas E. Lightburn, PhD ’11.
Tan said it is no accident that the process so closely resembles natural photosynthesis, as chemists are constantly drawing inspiration from nature in their work.
“Researchers in my field are always drawing inspiration from nature,” said Tan. “You take the basic lessons and you try to do it in an artificial way. In this work, we’re trying to learn lessons from nature, although we can’t copy nature directly.”
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For more info, contact: 
Ed Hayward
ed.hayward@bc.edu
617-552-4826 

Why chronic pain is all in your head (Neuroscience)



First study to show early brain changes predict which patients develop chronic pain

When people have similar injuries, why do some end up with chronic pain while others recover and are pain free? The first longitudinal brain imaging study to track participants with a new back injury has found the chronic pain is all in their heads –- quite literally.

A new Northwestern Medicine study shows for the first time that chronic pain develops the more two sections of the brain — related to emotional and motivational behavior — talk to each other. The more they communicate, the greater the chance a patient will develop chronic pain.

Chronic pain is one of the most expensive health care conditions in the U. S. yet there still is not a scientifically validated therapy for this condition.
The finding provides a new direction for developing therapies to treat intractable pain, which affects 30 to 40 million adults in the United States.
Researchers were able to predict, with 85 percent accuracy at the beginning of the study, which participants would go on to develop chronic pain based on the level of interaction between the frontal cortex and the nucleus accumbens.
The study is published in the journal Nature Neuroscience.
“For the first time we can explain why people who may have the exact same initial pain either go on to recover or develop chronic pain,” said A. Vania Apakarian, senior author of the paper and professor of physiology at Northwestern University Feinberg School of Medicine.
“The injury by itself is not enough to explain the ongoing pain. It has to do with the injury combined with the state of the brain. This finding is the culmination of 10 years of our research.”
The more emotionally the brain reacts to the initial injury, the more likely the pain will persist after the injury has healed. “It may be that these sections of the brain are more excited to begin with in certain individuals, or there may be genetic and environmental influences that predispose these brain regions to interact at an excitable level,” Apkarian said.
The nucleus accumbens is an important center for teaching the rest of the brain how to evaluate and react to the outside world, Apkarian noted, and this brain region may use the pain signal to teach the rest of the brain to develop chronic pain.
“Now we hope to develop new therapies for treatment based on this finding,” Apkarian added.
Chronic pain participants in the study also lost gray matter density, which is likely linked to fewer synaptic connections or neuronal and glial shrinkage, Apkarian said. Brain synapses are essential for communication between neurons.
“Chronic pain is one of the most expensive health care conditions in the U. S. yet there still is not a scientifically validated therapy for this condition,” Apkarian said. Chronic pain costs an estimated $600 billion a year, according to a 2011 National Academy of Sciences report. Back pain is the most prevalent chronic pain condition.
A total of 40 participants who had an episode of back pain that lasted four to 16 weeks — but with no prior history of back pain — were studied. All subjects were diagnosed with back pain by a clinician. Brain scans were conducted on each participant at study entry and for three more visits during one year.
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Other Northwestern authors on the paper include lead author Marwan N. Baliki, Bogdan Petre, Souraya Torbey, Kristina M. Herrmann, Lejian Huang and Thomas J. Schnitzer.
The study was funded by the National Institute of Neurological Disorders and Stroke of the National Institutes of Health grant NS35115.